Researching the genetics of beauty I noticed that ceramides, basically skin lipids, are orally active at improving beauty Further research suggests that some ceramides particular forms of which are described as sphingolipids are part of the composition of Myelin as well as metabolized to phosphocholine. The combination of a beauty lipid that is processed not onlyat the skin yet also at the CNS, with particular genes that supplies choline to the body is Longevity Beauty Nootropic area
Oral ceramides are published at improving dermal hydration as well as structure. Creating a probiotic that produces these ceramides improves population appearance. Noting that ceramides are lipids Noting that some lips like DHA are more beneficial than other lipids scientists should find the most physiologically beneficial ceramides to have the probiotics make possibly C16 DHAlike novel ceramides Some published beneficial ceramides are C18 while DHA is C16 At sphingolipids which look something like half a spermidine, It is possible the NH2 as well as OH groups of the more effective beauty ceramides placed on a C16 alkane like molecule could produce a beauty DHA Thus it is possible a beautifying pill could have cardiovascular or cognitive benefits. Also finding the most beautiful most youthful genotype of ceramide production describes skin beauty as well as youthfulness genes
Some ceramide genes are sphingomyelinases It is possible that upregulating the preferred human genetic versions of the ceramide sphingolipids could also effect improvements to myelin possibly improving cognition or CNS durability The complimentary papers at http://www.ncbi.nlm....les/PMC3477085/ as well as http://www.ncbi.nlm....les/PMC3338701/ Notes that Acid sphingomyelinase (ASM, EC 3.1.4.12) is a glycoprotein localised primarily to the lysosome where it catalyses the breakdown of sphingomyelin to ceramide and phosphorylcholine
One nifty thing about this is that apparently particular sphingolipids tell the body to upregulate beneficial dermal genetics. The paper suggests that small amounts of certain lipids tell the body to make larger amounts of beneficial lipids.
I urge people to figure out if there is a particular C16 DHAlike variant of sphingolipid from plants that has beneficial effects. People with personal genomics data may like visiting the paper to see what kind of sphingolipid activation genes like ELOVL genes and PPAR genes they would prefer to give their kids.
The pmc 3477085 paper notes that
The dynamic balance between these bioactive lipids is influenced by the activities of acid ceramidase, sphingosine kinase, sphingosine-1-phosphate phosphatase, sphingosine-acetyltransferase and ASM (acid sphingomyelinase). Among these enzymes, ASM holds a prominent position because it generates the first bioactive molecule in the rheostat. Consequently, abnormal ASM activity is linked to different pathological conditions. Dramatically decreased ASM (is crummy) thus the preferred beauty genes could also be cognitive durability genes.
