32 replies to this topic

[John250]

THC exhibited neuroprotective effect against all toxins probably by activation of PPAR-γ receptors
THC is an PPAR-γ agonist...

https://www.ncbi.nlm...les/PMC1892413/

https://www.ncbi.nlm...ubmed/10863546/

https://www.scienced...30530132531.htm

[Ruth]

https://www.ncbi.nlm...les/PMC1892413/

https://www.ncbi.nlm...ubmed/10863546/

https://www.scienced...30530132531.htm

"The cannabinoids namely, Δ9-THC (1), CBN (2) and a non-cannabinoid canniflavin-A (3) significantly induced NGF-stimulated neuritic outgrowth in the NS-1 cells. Canniflavin-A was observed to be the most potent among the compounds tested. Understanding the mechanism for neurotrophic action of these compounds and identification of target signal transduction pathways shall be important in their further evaluation as potential neurotrophic agents and their potential application in treatment of neurodegenerative diseases.(2017)"

[Ruth]

"curcumin stimulates NGF and the cannabinoids stimulate the NGF potentiation..

Omega 3 deficiencies make cb1/cb2?/ signaling aberrant.. and leads to decreased cb1 stimulation....

Curcumin-THC would be a powerful medicine in the terms of absorption and therapeutics uses...

Curcumin-THC proteins?

both stimulate cb1 and low doses lead to NDMA receptor antagonist affects thus lead to enhanced memory and also both active the VDR and Vitamin D liposome gel with magnesium would increase the "priming effect" of the various entourages effects from lemons and phenolics polyphenols, exercise,....psychedelics even activate the CB receptors....

Edited by Ruth, 17 October 2018 - 03:24 AM.