Yes it is possible that artificially increased levels of monoamine may incite "simulated" stress responses and can contribute to un-necessary anxiety by means of increasing cortisol, ACTH and other stress hormones. Peripherally noradrenaline can induce muscle contraction, constrict or dilate the pupil, depending on the receptor, it also speeds or reduces heart rate, depending on receptor. Noradrenaline causes peripheral and endothelial vasocontstriction, and by beta-adrenergic receptors it promotes positive inotropism and increased heart rate. By alpha-1-receptors NE will REDUCE heart rate, and increase BP.
Dopamine functions to control peripheral contraction in a manner generally opposite of noradrenaline - and thus why AntiParkinsons drugs that eliminate tremors are pro-dopamine drugs. Dopamine enhances GABA and depresses the nervous system by D2,D3,D4 receptors - whereas by D1 and D5 it also potentiates GABA but also increases thyroid hormone and second messengers.
Most of what it comes down to - are whether the NT in question is positively or negatively coupled to adenylate cyclase....and this then corresponds to calcium channels which regulate contraction and and constriction etc.
Depression can result from calcium overload and deficiency, but deficiency correlates with serotonin toxicity and results in a relative lack of emotional capacity.
Likewise, Histamine and Glutamate facilitate Ca2+ and cAMP the MOST - and this corresponds with increased emotions (in general).
Thus overemotional people commonly have either very high histamine or glutamate.