110 replies to this topic

[Walter Derzko]

 My guess is in the next 3-5 years arthritis will be a thing of the past, and good riddance. Nano tech be it C60-OO or graphene, or other nano substances, looks to really rock our world in a very very good way, in the next few years.

Why wait a few years, you can eliminate arthritis now, but most doctors don't want to. They are happy to have you come back regularly and just treat the symptoms.

[smccomas01]

With all do respect you are incorrect at least about RA. RA is an auto immune disease where the body, for what ever reason see something wrong and begins to over produce cytokines to attack the joints. One of the many differences is it is not just 1 joint or 1 side of the body, my very first flare started out in my feet and my left wrist. Later right ankle left knee. At times fingers swollen to the point that I could not make a fist, and then they were fine.

 

Then you have the other things that go on, the soul sucking fatigue that no matter how much sleep you get always tired. I once slept for 14 hours even then it was a struggle to get up. I literally watched my muscles atrophy, I went from 165ish to 146 in less than a year and most of that was muscle.

 

Now as far as what causes RA, my SWAG (scientific wild ass guess) is there is not one root cause. Some people report the onset right after a vaccine, some stressful event others some type of surgery. The meds specifically the biologic s address different cytokines and process's Enbrel and Humera  TNF-a, Orencia CD80 and CD86. With all of our advancement we still do not have a test that can identify specifically what bio markers are involved with the inflammation per specific individual. That would be why it is a trial and error method of treatment for RA.

 

So this statement that RA could be eliminated right now, no not even close can treat symptoms however root cause is still unknown.   

[free10]

I agree no cure for RA right now, just treatments and many of those fail over time. I would not recommend bed rest for it, as I know AS flares up usually at rest and you stiffen up and seems to be less of a problem on the go. The problem is many on the go with AS once they stop or pause even for a few minutes will get hit then with pains/flair ups, so they associate moving with making it worse, when it really seems to be the resting (sitting, laying down, or standing in one place basically that makes it worse and when my flare ups happened usually. I have had AS since my late teens.

 

Now one thing is the tiredness mentally and physically that comes with RA. It can be a little depressing LOL I do think the cure is not hat far off. One reason is I think aging, which they also are clueless about will be reversed and even cured in the next few years, and most non viral, mold,and bacterial diseases are in fact just aging, as their root cause. So cure it and the AS and most other things go away and stay away. The second reason I think it will be cured is rapidly advancing nano technologies currently, and, in the next few years. These 2 things will probably knock it out, and if they don't they will knock out 99% of the problems we have now, and leaving only this and a few more things to study and fix. Food, fresh water, super clean air, and energy should be abundant, along with the cures for "diseases". Fairy tales, pie in the sky, and insanity, yes, and in the labs or on the drawing boards now, and relatively few know or see this.

 

C60 is a form of this new tech. Nano tubes, graphene, and many others are as well, as we head towards the quantum reality, or very very close to it.

 

 

[Walter Derzko]

With all do respect you are incorrect at least about RA. RA is an auto immune disease where the body, for what ever reason see something wrong and begins to over produce cytokines to attack the joints. [..] 
So this statement that RA could be eliminated right now, no not even close can treat symptoms however root cause is still unknown.

Vets working with animals in the last 60 years have cured most chronic human diseases in animals, including RA. When did you last see a cow or a pig with RA? ROOT CAUSE: It's an essential trace mineral deficiency. Trace microminerals are needed as cofactors for most enzymes in the body to work properly. Mineral soil depletion in the last 100 years has led to the increase in most chronic human diseases and mortality rates that were not even on the map 100 years ago. Do your homework and don't listen to most conventional doctors. They only know what the pharma industry tells them and they are not interested in a cure. Check the medical literature and clinical studies. PUBMED is your friend. http://www.ncbi.nlm.nih.gov/pubmed

Attached Files

•  trace minerals.png   50.18KB   2 downloads
•  minerals-up-down.jpg   18.51KB   2 downloads

Edited by Walter Derzko, 07 April 2015 - 05:22 PM.

[smccomas01]

Free10 do you have RA if so did you notice an improvement when taking C60OO? 

[smccomas01]

Walter, respectfully you are incorrect. I think you are focusing on the "arthritis" part of the name which in my opinion was a mistake (years ago) to begin with. It really should be called Rheumatoid Auto Immune disorder. This is from a link related to RA and dogs however accurate for humans to. 

 

n rheumatoid arthritis, the body mistakes some of its own protein for foreign protein. It then makes antibodies against its own protein. These unique antibodies are collectively called 'rheumatoid factor.' The antibodies and protein form immune complexes, which are then deposited in the joint. These complexes trigger a process called inflammation, which is how the body reacts to injury. In the case of rheumatoid arthritis, the body tries to rid itself of the injurious immune complexes, but manages to only create more damage to the joint. The process then becomes self-perpetuating. Eventually, the cartilage and even the bone in the joint are worn away.

→ source (external link)

 

If it was as simple as mineral deficiency it would be cured. 

 

 

[Walter Derzko]

RA, autoimmunity and gut microbiome.

The appears to be a strong link between RA, autoimmunity and gut microbes that are influenced by diet and lack of trace micronutrients.


Walter Derzko

Front Microbiol. 2015 Jan 14;5:783. doi: 10.3389/fmicb.2014.00783. eCollection 2014.

Interaction between extracellular matrix molecules and microbial pathogens: evidence for the missing link in autoimmunity with rheumatoid arthritis as a disease model.

Sofat N1, Wait R2, Robertson SD1, Baines DL1, Baker EH1.


Abstract

Rheumatoid arthritis (RA) is an autoimmune disease characterized by inflammation followed by tissue rebuilding or fibrosis. A failure by the body to regulate inflammation effectively is one of the hallmarks of RA. The interaction between the external environment and the human host plays an important role in the development of autoimmunity. In RA, the observation of anti-cyclic citrullinated peptide antibodies (ACPA) to autoantigens is well recognized. Citrullination is a post-translational modification mediated by peptidyl arginine deiminases, which exist in both mammalian and bacterial forms. Previous studies have shown how proteins expressed in the human extracellular matrix (ECM) acquire properties of damage-associated molecular patterns (DAMPs) in RA and include collagens, tenascin-C, and fibronectin (FN). ECM DAMPs can further potentiate tissue damage in RA. Recent work has shown that citrullination in RA occurs at mucosal sites, including the oral cavity and lung. Mucosal sites have been linked with bacterial infection, e.g., periodontal disease, where exogenous pathogens are implicated in the development of autoimmunity via an infectious trigger. Proteases produced at mucosal sites, both by bacteria and the human host, can induce the release of ECM DAMPs, thereby revealing neoepitopes which can be citrullinated and lead to an autoantibody response with further production of ACPA. In this perspectives article, the evidence for the interplay between the ECM and bacteria at human mucosal surfaces, which can become a focus for citrullination and the development of autoimmunity, is explored. Specific examples, with reference to collagen, fibrinogen, and FN, are discussed.


KEYWORDS:

citrullination; extracellular matrix; infection; lung; microbiome; periodontal disease; rheumatoid arthritis


###

Rheumatology (Oxford). 2014 Dec 23. pii: keu469. [Epub ahead of print]
Gene, environment, microbiome and mucosal immune tolerance in rheumatoid arthritis.
Catrina AI1, Deane KD2, Scher JU2.
Author information
• 1Rheumatology Unit, Department of Medicine, Karolinska University Hospital and Institutet, Stockholm, Sweden, Division of Rheumatology, University of Colorado, School of Medicine, Aurora, CO and Division of Rheumatology, Department of Medicine, New York University School of Medicine and Hospital for Joint Diseases, New York, NY, USA. anca.catrina@ki.se.
• 2Rheumatology Unit, Department of Medicine, Karolinska University Hospital and Institutet, Stockholm, Sweden, Division of Rheumatology, University of Colorado, School of Medicine, Aurora, CO and Division of Rheumatology, Department of Medicine, New York University School of Medicine and Hospital for Joint Diseases, New York, NY, USA.
Abstract
RA is a complex multifactorial chronic disease that transitions through several stages. Multiple studies now support that there is a prolonged phase in early RA development during which there is serum elevation of RA-related autoantibodies including RF and ACPAs in the absence of clinically evident synovitis. This suggests that RA pathogenesis might originate in an extra-articular location, which we hypothesize is a mucosal site. In discussing this hypothesis, we will present herein the current understanding of mucosal immunology, including a discussion about the generation of autoimmune responses at these surfaces. We will also examine how other factors such as genes, microbes and other environmental toxins (including tobacco smoke) could influence the triggering of autoimmunity at mucosal sites and eventually systemic organ disease. We will also propose a research agenda to improve our understanding of the role of mucosal inflammation in the development of RA.
© The Author 2014. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oup.com.
KEYWORDS:
break in tolerance; environmental risk factors; genetic susceptibility; microbiome; rheumatoid arthritis

[smccomas01]

I have read that before they have also found a link again in some cases between oral health and the onset of RA. Interesting stuff do not want to go to far off topic though what I can say is C60 OO has helped me considerably. What would be a good topic is the why my hypothesis would be its anti oxidant house cleaning action.  

[Walter Derzko]

C-Sections, Gut microbiota and subsequent diseases


External influence of early childhood establishment of gut microbiota and subsequent health implications.
Front Pediatr. 2014 Oct 9;2:109. doi: 10.3389/fped.2014.00109. eCollection 2014.
Munyaka PM1, Khafipour E2, Ghia JE3.

Author information


Abstract

Postnatal maturation of immune regulation is largely driven by exposure to microbes. The gastrointestinal tract is the largest source of microbial exposure, as the human gut microbiome contains up to 10(14) bacteria, which is 10 times the number of cells in the human body. Several studies in recent years have shown differences in the composition of the gut microbiota in children who are exposed to different conditions before, during, and early after birth. A number of maternal factors are responsible for the establishment and colonization of gut microbiota in infants, such as the conditions surrounding the prenatal period, time and mode of delivery, diet, mother's age, BMI, smoking status, household milieu, socioeconomic status, breastfeeding and antibiotic use, as well as other environmental factors that have profound effects on the microbiota and on immunoregulation during early life. Early exposures impacting the intestinal microbiota are associated with the development of childhood diseases that may persist to adulthood such as asthma, allergic disorders (atopic dermatitis, rhinitis), chronic immune-mediated inflammatory diseases, type 1 diabetes, obesity, and eczema. This overview highlights some of the exposures during the pre- and postnatal time periods that are key in the colonization and development of the gastrointestinal microbiota of infants as well as some of the diseases or disorders that occur due to the pattern of initial gut colonization.


KEYWORDS:

antibiotics; cesarean section; diet; gut microbiota; immunity; inflammatory diseases

[Astroid]

Have you researched  rheumatoid arthritis + LDN (Low Dose Naltrexone) ?  

 

It stopped severe hives I had for 8 years.. breaking out 3 times a week at the end.. and the Doctors never figured out any part of the problem.  I take 4.5 mg a night.. Plus 600 mg of Alpha Lipoic Acid a day.. and the hives stopped immediately. That has been 4+ years ago.

 

LDN optimized your immunity system, it does not super charge it.. I consider autoimmune problems like having water in your gas tank.. the body is misfiring.  There are no side effects from LDN. Adults take from 3.0 to 4.5 mg a night. Morphine will not work while on LDN.

 

Numerous items come up if you search for it. For $1/ day.. or less from Skip's Pharmacy in Boca Raton.. the largest provider in the USA.. Skip has MS and on youtube says without this he would be in a wheelchair.

 

Attached are some articles.   

 

Dr Burt Berkson complete LDN talk & Q&A 

 

The MD who discovered working with AIDs patients how to increase your immunity 300%

 

 

1 hour 31 minutes long.

 

 

 

http://www.nytimes.c...tis-part-1.html

 

Quote 

Q. I was given a diagnosis in December 2012 of mild-mod R.A., and started methotrexate in January 2013. In June I stopped methotrexate and began taking low-dose naltrexone (LDN). I discovered LDN on the Web after searching for infectious causes of R.A. I had heard an interview with Dr. Ewald on the People’s Pharmacy radio show about infections as the cause of some cancers and autoimmune disorders. It made perfect sense to me and reflected my experience (sudden onset of full blown RA after sinus infections and flu vaccine). I now take 4.5 milligrams of LDN per day and I feel better than I have in years. If I feel stiffness or pain late in the day ibuprofen helps but I rarely need it. LDN works by supporting the immune system and increasing the availability of endorphins. It is safe and F.D.A. approved (opiate addiction) at 50 milligrams per day (the max dose for R.A. is 4.5 milligrams per day). I try not to be cynical but it’s hard not to believe that big pharma plays a role in why this drug is not well known in the medical world.

 

 

Attached Files

•  Bernard Bihari, MD - LDN Interview 9-23-2003.pdf   236.8KB   0 downloads
•  ldn_for_disease_prevention_quality_of_life.pdf   149.74KB   0 downloads
•  Bernard Bihari, MD - History.doc   45.5KB   0 downloads
•  LDN - RARE Drug Actually Helps Body Heal Itself.doc   46.5KB   1 downloads

[free10]

Free10 do you have RA if so did you notice an improvement when taking C60OO? 

 

I have AS, which is considered a form of RA. Now most of the problems with my AS stopped many decades ago, as far as pain. Started in my hips, then went to my spine, then my eye/eyes (iritis), but with that said I did develop a problem in my knees maybe 15 years ago right above the knee caps, and triggered usually by climbing a ladder or kneeling/squatting down. Now was this AS don't know, but I do know the pain would stop in them, even if climbing a ladder within a few hours of my first dose of C60. Go enough days without anymore C60, and I start to notice it again.

 

Is it AS and why it goes away with C60 is unknown, and C60 can't fix it all. For a few days I had very bad pain, after trying to lift some weights again and nothing took it away. Not aspirin, prednesone, C60. Then I got introduced to a bottle of Thermaflex liniment gel, and within a few minutes of her applying it to that shoulder, for the first time in days it eased and then stopped, and stayed stopped for hours. When it came back I would just rub another small dab in. MSM seems to be why in worked.

 

I have seen some amazing things from C60, such as stopping the flu cold in a few hours, burning my hands with a torch and not suffering from a lot of damage from it, and of course the knees. I have  no way to know, if C60 is doing a lot more for me.

[Walter Derzko]

Rheumatoid Arthritis and Mycoplasma.
Turns out that RA is triggered by mycoplasma bacteria. Take at two week course of the tetracycline derivative for mycoplasma and your RA should be gone. Then you need to take the micronutrients and trace minerals to repair the damage done by the bacteria in your joints .There are 264 papers on PUBMED http://www.ncbi.nlm....itis mycoplasma

Here are four random paper pointing to mycoplasma

###

Lik Sprava. 2005 Jul-Sep;(5-6):28-32.

[Frequency of different infectious agents persistence in mononuclear leukocytes of blood and synovial fluid in patients with rheumatoid arthritis].

[Article in Ukrainian]

Petrov AV.

Abstract

The study of persistence in mononuclear leukocytes (ML) of blood and synovial fluid of 218 patients with rheumatoid arthritis (RA) Cytornegalovirus (CMV), the 1-st and 2-nd types of Herpes virus simplex (VH), Epstain-Barr virus (VEB), Mycoplasma arthritidis (Ma), Mycoplasma fermentans (Mf), Ureaplasma urealiticum (U), Chlamidia trachomatis (Ct), viruses of Hepatitis B and C was carry out by direct and indirect immunofruorescence, immunoenzymatic analysis and polymerase chain reaction. An increased frequency of contamination of blood ML with infectious agents in patients with RA was established (57,4% compared with 16,7% in control group). The following infectious agents were revieled more frequently: in ML of blood and synovial fluid the Ma (relatively 20,5% and 15,9%), Mf (15,6% and 13,2%), Ct (18,4% and 13,2%), VH (27,1% and 10,5%), VEB (12,7% and 5,3%) and CMV (11,2% and 7,9%). Types of frequency dynamics of ML contamination with these infectious agents in different time phases of RA were determined.


###

Biochem Biophys Res Commun. 2008 May 2;369(2):561-6. doi: 10.1016/j.bbrc.2008.02.079. Epub 2008 Feb 26.

Mycoplasma fermentans glycolipid-antigen as a pathogen of rheumatoid arthritis.

Kawahito Y1, Ichinose S, Sano H, Tsubouchi Y, Kohno M, Yoshikawa T, Tokunaga D, Hojo T, Harasawa R, Nakano T, Matsuda K.

Author information
Abstract

Mycoplasma fermentans has been suspected as one of the causative pathogenic microorganisms of rheumatoid arthritis (RA) however, the pathogenic mechanism is still unclear. We, previously, reported that glycolipid-antigens (GGPL-I and III) are the major antigens of M. fermentans. Monoclonal antibody against the GGPL-III could detect the existence of the GGPL-III antigens in synovial tissues from RA patients. GGPL-III antigens were detected in 38.1% (32/84) of RA patient's tissues, but not in osteoarthritis (OA) and normal synovial tissues. Immunoelectron microscopy revealed that a part of GGPL-III antigens are located at endoplasmic reticulum. GGPL-III significantly induced TNF-alpha and IL-6 production from peripheral blood mononulear cells, and also proliferation of synovial fibroblasts. Further study is necessary to prove that M. fermentans is a causative microorganism of RA; however, the new mechanisms of disease pathogenesis provides hope for the development of effective and safe immunotherapeutic strategies based on the lipid-antigen, GGPL-III, in the near future.

###


J Clin Microbiol. 2007 Mar;45(3):953-7. Epub 2006 Nov 22.

Distribution of Mycoplasma pneumoniae and Mycoplasma salivarium in the synovial fluid of arthritis patients.

Johnson SM1, Bruckner F, Collins D.

Author information


Abstract

By use of a very sensitive nested PCR method targeting part of the strongly conserved mycoplasmal 16S RNA genes, Mycoplasma pneumoniae was found in the synovial fluid of 19/24 (79%) of rheumatoid arthritis patients, 6/6 (100%) of patients with nonrheumatoid inflammatory arthritis, and 8/10 (80%) of osteoarthritis patients attending the rheumatology clinic for drainage of joint effusions. It was not found in the synovial exudates of 13 people attending the orthopedic clinic with traumatic knee injuries or undergoing surgery for knee replacement. However, M. pneumoniae was detected in 2/4 synovial biopsy specimens from orthopedic patients with traumatic knee injuries. M. pneumoniae was associated with the increased synovial fluids found in arthritic flares but was not found in the synovial fluids of trauma patients. Mycoplasma salivarium occurred sporadically. Mycoplasma fermentans had previously been isolated from patients with inflammatory cellular infiltrates, such as rheumatoid arthritis, but it was not detected for osteoarthritic patients from either clinic. It is possible that these organisms may contribute to chronic inflammation within the joints.

###

Scand J Rheumatol. 2006 Nov-Dec;35(6):459-62.

Potential triggering infections of reactive arthritis.

Butrimiene I1, Ranceva J, Griskevicius A.

Author information

Abstract

OBJECTIVES:

The aim of the study was to investigate possible triggering infections causing reactive arthritis (ReA) of urogenital origin.

METHODS:

One hundred and twenty ReA patients, 85 control group patients with other arthritides (61 with rheumatoid arthritis, 13 with osteoarthritis, and 11 with microcrystal arthritis), and 52 healthy persons were tested for urogenital tract inflammation and several infectious agents. Ligase chain reaction was used for detection of Chlamydia trachomatis (CT). Genital mycoplasmas Ureaplasma urealyticum (Uu) and Mycoplasma hominis (Mh) were tested using the Mycoplasma Duo Test (MDT). Only titres greater than 10(4) CCU/mL were accepted as pathogenecity threshold levels for Uu.

RESULTS:

Inflammation of the urogenital tract (most frequently urethritis in men and cervicitis in women) was found in 95% of patients with acute ReA. Possible causative pathogens were identified in 58% of ReA patients. CT was found in 29%, Uu in 21%, and Mh in 8% of patients with ReA. While CT and Uu were found more often in HLA-B27-positive than in HLA-B27-negative patients, this was statistically proved only for CT. In ReA males Uu was found four times more frequently than in men with other arthritides.

CONCLUSIONS:

In active ReA of urogenital origin, inflammation of the urogenital tract is found in the majority of patients. Although CT is the main microorganism associated with urethritis in men and cervicitis in women, mycoplasmas, especially Uu, may be possible aetiological factors for ReA.

[niner]

Reactive arthritis has been known for a long time, but has been underdiagnosed, imho.   It's caused by a sub-clinical infection that presents an antigen that looks like your own tissue.  The immune system is activated, but it attacks you along with the microbe.   I had it once, and it was misdiagnosed repeatedly.  It was caused by bad clams.  (Seriously.  Sounds like a punchline or something, but I got a GI infection from eating clams, or at least got it the appropriate length of time after eating them, and the ReA followed within a short time.   It was never correctly diagnosed, but a course of Biaxin for an unrelated condition cured it.) It sounds like from what Walter just posted that a lot of RA is actually ReA.  A lot but not all, or even most.   It would certainly be worth looking for immunological or other markers of infection if you have RA, or maybe just try a course of antibiotics.  If it's mycoplasma, you would probably need a long course of a macrolide.  It's hard to kill.

[nowayout]

I have AS, which is considered a form of RA.

 

I also have been diagnosed with A.S.  Ankylosing Spondylitis and Rheumatoid arthritis are both autoimmune/inflammatory conditions and some of the same drugs are used to treat both, but the current medical consensus is that they are quite different diseases with very different clinical symptoms and etiologies.    Their effects on bones and connective tissue are very different.   Some drugs that work for RA don't work for AS at all. 

[free10]

 

I have AS, which is considered a form of RA.

 

I also have been diagnosed with A.S.  Ankylosing Spondylitis and Rheumatoid arthritis are both autoimmune/inflammatory conditions and some of the same drugs are used to treat both, but the current medical consensus is that they are quite different diseases with very different clinical symptoms and etiologies.    Their effects on bones and connective tissue are very different.   Some drugs that work for RA don't work for AS at all. 

 

 

I had mu first syptoms from in in my late teens, and more syptoms by my mid 20s, and was diagnosed around the age of 40, by an xray technician looking at my back xrays. I have zero faith in medical after a life time of experience with them, and have done my best to take care of it myself.

 

I have used the internet a lot, since the late 90s to learn more and study any possible solutions or help they might offer. I noticed many of the drugs have bad side effects such as increases in cancers, and others worked only for a short period, and then it came back with a vengence. I would rather read about it then be one that goes through this first hand.

 

Most of the pain I have suffered with it has stopped, but the damage is there  and I suspect continuing at some level. Here are some facts I have run across than most will not know or be told. Hip pain in the teens to early 20s is a fairly common first sign you have it. It seems to be from the sacroiliac joints. COPD is also a side effect for many and smoking does not cause nor make the COPD or fibrosis in the lung worse. Iritis of the eyes is also common from it. It can hit the heart valves as well. Keep physically moving limits the pain and inflammations that occur. It is very nasty.

 

I see it as a product of aging cells making errors somewhere along the line. And see reversing aging, and repair or regeneration of the damage as the cure. I think C60-00 should help both pain and damage protection over time.

 

It would be interesting, if I was younger with the different and more often pain and problems, to test this hypotheis on myself, just to see. Now it is more sporadiac and harder to tell. I know the knee pain went away as long as I take some every so may day, but don't know if that is AS or some other cause/problem. I am getting closer to 70 all the time LOL

[fntms]

Rheumatoid arthritis is considered to be an auto-immune syndrome. It has recently been successfully treated with n-acetyl glucosamine (not regular glucosamine!). A starting dose is probably about 2g twice a day.

See these references:
http://www.newscient...=mg19426074.500
http://www.ncbi.nlm....pubmed/22434264

n-acetyl glucosamine also treats multiple sclerosis and other auto-immune diseases.
http://www.scienceda...10930123057.htm

As an update I have been taking a moderate dose (2 caps daily) of jarrow's NAG for a while and my rheumatisms (right toe, left foot) have mostly disappeared. I also added 120mg ginkgo extract at the same moment which probably helps as well.
Now for my lasting neck pain, not caused by rheumatisms, nothing seems to really work apart from tramadol

[smccomas01]

I am glad to hear that not about the neck about the other issues. Hopefully you will not experience the full on flare. 

[stefan_001]

I am glad to hear that not about the neck about the other issues. Hopefully you will not experience the full on flare. 

 

Hello hope you are doing fine. Would appreciate to hear how it is going with the C60oo and NAG for your RA. I am thinking what to recommend to my 76 year old mom who is increasingly struggling with arthritis. Experiences from others welcome too.

Stefan

[smithx]

The great thing about NAG (n-acetyl glucosamine) is that even if the problem is actually osteoarthritis, it would still help because it is, after all, glucosamine and can be used to produce hyaluronic acid which lubricates the joints.

 

 

 

I am glad to hear that not about the neck about the other issues. Hopefully you will not experience the full on flare. 

 

Hello hope you are doing fine. Would appreciate to hear how it is going with the C60oo and NAG for your RA. I am thinking what to recommend to my 76 year old mom who is increasingly struggling with arthritis. Experiences from others welcome too.

Stefan

 

 

[Astroid]

14% Gallium Nitrate (galliumnitrate.com) kills inflammation and thus joint pain ! Works wonders where the skin is thin.. knees, ankles, feet, hands, elbows and yes the neck. 43% is used for horse's limbs and knees. Nothing is harder on knees than a race horse.  Last year I applied a capful daily to my knee after still having pain 3 weeks following synthetic fluid injections.  In 3 days 50% of the pain was gone. I applied it daily for 3 weeks and then only as needed. I applied it maybe 10 times over the next year.. and was active with painting and raising newborn german shepherd pups. No pain resulted.   

 

Also 1 teaspoon of MSM-Sulfur in a shot of water 2 x day helps one detox.. Sulfur is in every cell membrane of the body. A horse vet confirmed they give it to race horses.. to detox them. It allows water and oxygen to exchange between the cell walls.  It helped mine and others back and joint pains.  I found a 10 lb bucket on amazon for some $36 and free shipping !  One can see results in 1-2 days. Yes, you have a Sulfur mineral deficiency !  It is said to be the 5th most important element in your body.. and the 4th most abundant mineral in the body.  

 

Soaking in Epsom Salt (Magnesium) also reduces joint pain.

 

Carbon 60 makes me feel great waking up. I can not really tell it's result for inflammation however. I am sure it works for that however.

 

Arthritis is a immunity problem. Low Dose Naltrexone (LDN) optimizes the immunity and stops the progression.    

14% Gallium Nitrate (galliumnitrate.com) kills inflammation and thus joint pain ! Works wonders where the skin is thin.. knees, ankles, feet, hands, elbows and yes the neck. 43% is used for horse's limbs and knees. Nothing is harder on knees than a race horse.  Last year I applied a capful daily to my knee after still having pain 3 weeks following synthetic fluid injections.  In 3 days 50% of the pain was gone. I applied it daily for 3 weeks and then only as needed. I applied it maybe 10 times over the next year.. and was active with painting and raising newborn german shepherd pups. No pain resulted.   

 

Also 1 teaspoon of MSM-Sulfur in a shot of water 2 x day helps one detox.. Sulfur is in every cell membrane of the body. A horse vet confirmed they give it to race horses.. to detox them. It allows water and oxygen to exchange between the cell walls.  It helped mine and others back and joint pains.  I found a 10 lb bucket on amazon for some $36 and free shipping !  One can see results in 1-2 days. Yes, you have a Sulfur mineral deficiency !  It is said to be the 5th most important element in your body.. and the 4th most abundant mineral in the body.  

 

Soaking in Epsom Salt (Magnesium) also reduces joint pain.

 

Carbon 60 makes me feel great waking up. I can not really tell it's result for inflammation however. I am sure it works for that however.

 

Arthritis is a immunity problem. Low Dose Naltrexone (LDN) optimizes the immunity and stops the progression.    

[jeanlzt11]

Hello,  I started using C60 about 3 weeks ago and noticed the arthritic finger joint I've had for the past year or so doesn't hurt as much anymore.  I also rub a little right on the joint.  I wondered if anyone else has noticed this?  I only have arthritis in one finger so I don't know if it's just co-incidence.

Thanks