3 replies to this topic

[YOLF]

Met someone with mito trifunctional protein deficiency and I'm wondering how, as a Mito OA, how C60 might affect someone who acquired this as an adult? People with this aren't able to convert long chain fatty acids into energy and develop a protein deficiency.

 

I'm thinking that this along with maybe mitochondrial uncoupling could lead to improvements or maybe even be corrective to the condition?

 

What are the unaccounted for fats in EVOO? IIRC, it's mostly O6 FAs? I take it that the long chains are O9 FAs and EVOO shouldn't have much if any of those? What about the C60 "razor oil" at RevGenetics?

Edited by YOLF, 19 November 2015 - 05:02 PM.

[niner]

This is a genetic disease of long chain fatty acid metabolism.  C60oo wouldn't be a good choice for them, as they shouldn't be consuming long chain fatty acids.  Olive oil is almost entirely long chain fatty acids.  The problem these people have is that heart and skeletal muscles like to eat long chain fatty acids.  Since this case is adult onset, they probably have a milder version, and might be able to handle some fatty acids.  I wonder if short chain fatty acids could be a substitute fuel for them?  Since it's a problem of energy conversion, they might well be helped by a mitochondrial antioxidant.  I'd probably choose MitoQ in this case.   Only if they can safely handle small amounts of vegetable oils would I consider c60oo.  If they try either of these, let us know how it turns out.

[YOLF]

Could C60 be attached to a short chain fatty acid? Any good candidates?

[Nuke]

Maybe a good use for MCT oil? I understood that Lauric acid acts like a long chain fatty acid when producing energy, and MCT should have very little to none of it.