1 reply to this topic

[Supierce]

https://www.scienced...022282818303614

 

• The NAD⁺ precursor nicotinamide mononucleotide (NMN) acutely stimulates cardiac glycolysis.
• During ischemia, NMN is cardioprotective via stimulating glycolytic ATP synthesis.
• When delivered at reperfusion, NMN is cardioprotective via enhancing acidosis.
• Acidosis induced by NMN may have implications for cancer and other phenotypes.

Edited by Supierce, 01 July 2018 - 01:31 PM.

[Phoebus]

 

 

Glycolysis (from glycose, an older term^[1] for glucose + -lysis degradation) is the metabolic pathway that converts glucoseC₆H₁₂O₆, into pyruvate, CH₃COCOO⁻ + H⁺. The free energy released in this process is used to form the high-energy molecules ATP (adenosine triphosphate) and NADH (reduced nicotinamide adenine dinucleotide).^[

 

 

so NMN supplementation indirectly raises both ATP and NADH

 

what the difference between raising NAD+ vs raising NADH? 

 

anyone understand this?