Hello,
I am on to an idea - but may need some help / more scientific minds to either realize it or falsify it. Please bare with me.
Biophotons Background:
Biophotons are generated within the metabolism of the brain during redox/radical processes. More specifically, biophotons originate from bioluminescent reactions of Reactive oxygen species (ROS) and Reactive Nitrogen Species (RNS). ROS and RNS are generated by metabolic reactions such as the mitochondrial respiration chain, lipid peroxidation, oxidation of catecholamines, ect. --> So in theory, biophotons are produced/regulated by the presence of ROS/RNS, which are in turn produced/regulated by mitocondrial oxidative metabolism (mostly) and few others.
Now the preface to the theory is that after a spike-related electrical signal travels across the neuron, the affected mitochondrias will create a spatio-temporal biophoton pattern. This is the case with re-representing a seen visual image within the conscious, and as well as visually "remembering" - as studies have shown that in the hippocampus, the intensity of biophotons production are in relation to the neural metabolic activity at hand. There is a theory on this which attends to how we consciously visualize something, but this is not where I am headed with this post.
Thick of it - these biophotons can be absorbed by natural photosensitive chromphores of the cells, more specifically within the inner membrane of the mitochondria, as these contain flavinic, pyridinic, and porphyrin rings. In the same instance, I will get back to this, but other photo-acceptors are created during regulated lipid peroxidation of membranes (which will bring me to Isochroma and his initial theory about high amounts of fish oil while taking piracetam, and how he thought it helped the neurons "split" much easier). --> These photosensitive molecules "can transfer the absorbed biophoton energy to nearby biomolecules via resonance energy transfer, which can induce conformation changes and trigger complex signal processes in cells." <--
The Idea: These flavin, pyridinic, and porphyrin rings (as well as the lipid peroxidation product) bring up an interesting aspect, if these photosensitive molecules act as the "catcher" to the regulated/organized "pitcher" of biophotons - wouldn't the rate limiting step between conscious visual thought and conformational cellular changes be these photosensitive molecules? (The pitcher can throw all he wants, but if there is no catcher, the game bottlenecks, does not progress, and is inherently unplayable.)
My Question here is: Does the piracetam molecule with it's pyrrolidine ring undergoe a metabolic process in which it either becomes photosensitive itself? or could it supplement towards the increased production/capacity of the photosensitive inner mitochondrial molecules?
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As Isochroma, and thru my own personal experience, have realized is that piracetam is hugely synergistic with large amounts of fish oil. Could this subjective effect be due to an increased production/capacity for photosensitive molecules due to the increased lipid membrane peroxidation rates thru the large amounts of supplemented fish oil? The summative increase in photosensitive molecules by the fish oil, the piracetam, and finally, the synergistic effect of piracetam on mitocondrial optimization leading to optimized metabolic/lipid peroxidation - would lead to increased biophoton production & increased biophoton sensitivity (two birds with one stone) - could this be the real reason behind piracetam's nootropic effects (which would downstream compliment piracetam's established nootropic mechanisms as well)??
Thank you for your time.
I hope this makes sense.
What do you think?
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