38 replies to this topic

[Shepard]

There are a few discussions on this topic at M&M/Avant if you search back a little ways.

[jaydfox]

Thanks cnorwood19 (sorry, don't recall your name, if I ever learned it).

The last paragraph calls a lot into question, as far as whether the exercise itself really burns so much more fat. There wasn't control for diet, and this doesn't take into account a possible shift into lipid-burning during rest (the other 23.5 hours of the day).

But, that doesn't invalidate the general premise, as far as trying to burn fat calories in general and lose subcutaneous (and possibly visceral?) fat. I myself am still fairly overweight, so this information could be useful to me in the coming months. (Regarding my overweightedness, I am officially no longer obese! Yay! 195 pounds at 174 cm... wait, can't mix standard and metric... anyway, BMI is 29.2.)

[Shepard]

How trained you are is going to play a part in this, too. (HIIT vs. endurance/aerobic)

[jaydfox]

It's the anti-oxidant production that makes you come out ahead, in terms of free radicals. Without the boost in anti-oxidants, you'd produce more free radicals (and sustain more damage from them) if you exercise, as opposed to not exercising. But the 30 minutes of increased free radicals is balanced out by the 23.5 hours of increased anti-oxidant activity.

In the long term (six decades, 8 decades, etc.), those free radicals will affect your lifespan potential, possibly by years (or possibly just months, hard to say how much free radicals factor into longevity).

In your view is it better to take the common antioxidants used by people here (lipoic acid, green tea, vit c and e, etc) or take none and exercise or lastly take them and exercise?

Sorry, I seem to have missed this (last comment on previous page). Anyway, I guess my position seems more clear now: the third option, take the anti-oxidants and exercise. But take the anti-oxidants after a workout, not before.

[Athanasios]

oh, something occured to me.... I take 3 capsules of AOR's alpha lipoic acid a day, which is time released over 8 to 10 hours, so it is 24/7. I was operating under the assumption that others were doing something similar.

I wasn't suggesting purposely spiking them for a short bit to prevent the exercise induced ROS.


Haha, sorry, I am a dunce today...not a lot of sleep cause I drove 19 hours yesterday.

- chris

[jmmathieu]

(Regarding my overweightedness, I am officially no longer obese! Yay! 195 pounds at 174 cm... wait, can't mix standard and metric... anyway, BMI is 29.2.)

Lol, the BMI is horrible though. My BMI is 29 too, but I only have 10% body fat. I'm 6'0 and 205 pounds. Anyway, I have to say this has been a really good thread and thanks to everyone for all the useful info. That stuff on HIIT looks great, I'm gonna check it out on PubMed now...

[Athanasios]

Yeah, the science of tabata workouts was from this study:

PMID: 9139179

Check out this thread on it:

http://www.imminst.o...=171&t=11449&s=

But instead of taking their word for it, try it once and see if it FEELS like it does what they claim...because boy, they whoop my ***

[doug123]

Thanks cnorwood19 (sorry, don't recall your name, if I ever learned it).

The last paragraph calls a lot into question, as far as whether the exercise itself really burns so much more fat. There wasn't control for diet, and this doesn't take into account a possible shift into lipid-burning during rest (the other 23.5 hours of the day).

But, that doesn't invalidate the general premise, as far as trying to burn fat calories in general and lose subcutaneous (and possibly visceral?) fat. I myself am still fairly overweight, so this information could be useful to me in the coming months. (Regarding my overweightedness, I am officially no longer obese! Yay! 195 pounds at 174 cm... wait, can't mix standard and metric... anyway, BMI is 29.2.)

Dude, when I was in rehab once in Azusa, California, I was 230 lbs. I literally had to fast for a few months to lose most of that weight. I should convert these terms to metric units.

[Shepard]

One more from the BR newsletter this week:

    Oxidative stress in marathon runners: interest of antioxidant supplementation.

        * Gomez-Cabrera MC,
        * Martinez A,
        * Santangelo G,
        * Pallardo FV,
        * Sastre J,
        * Vina J.

    Catholic University of Valencia, Spain.

    We have recently reported that xanthine oxidase is involved in the generation of free radicals in exhaustive exercise. Allopurinol, an inhibitor of xanthine oxidase, prevents it. The aim of the present work was to elucidate the role of exercise-derived reactive oxygen species in the cell signalling pathways involved in the adaptation to exercise in man. We have found that exercise causes an increase in the activity of plasma xanthine oxidase and an activation of NF-kappaB in peripheral blood lymphocytes after marathon running. This activation is dependent on free radical formation in exercise: treatment with allopurinol completely prevents it. In animal models, we previously showed that NF-kappaB activation induced by exhaustive physical exercise leads to an increase in the expression of superoxide dismutase, an enzyme involved in antioxidant defence. We report evidence in man that reactive oxygen species act as signals in exercise as decreasing their formation prevents activation of important signalling pathways which can cause useful adaptations in cells.

    PMID: 16923247 [PubMed - in process]