Thanks Mathias for recovering my post [ang]
originally posted by jackinbox (Jan 30 2007-21:03) :
could the dopamine increase be of any use, as an exemple, in case of ADHD? To avoid dopamine depletion when using others meds? I'm just wondering...
i dont think so, as adhd is not a problem of the ammount of dopamine
this is why moai's wont work for adhd either
slowing the re uptake of dopamine works
This statement isn't absolutely true-
# Arch Neurol 50: 286-288 (1993). Deprenyl in attention deficit associated with Tourette's syndrome. Jankovic J.
While central nervous system stimulants usually improve attention deficit hyperactivity disorder (ADHD) associated with Tourette's syndrome, they often exacerbate tics and can produce other potentially serious complications. Because deprenyl may have a stimulatory effect and monoamine oxidase inhibitors have been shown to ameliorate hyperactive behavior, we studied this drug in children with the Tourette's syndrome-ADHD combination. Twenty-nine patients, 25 boys and four girls, with a mean age of 11.2 years (range, 6 to 18 years) and duration of symptoms for an average of 6.2 years (range, 1 to 13 years), were enrolled in this open trial after they became refractory to conventional treatments for ADHD. The average duration of treatment with deprenyl was 6.7 months (range, 3 to 15 months) and the average daily dose was 8.1 mg/d (range, 5 to 15 mg/dL). Twenty-six of all patients (90%) reported clinically meaningful improvement in their ADHD (score > or = 2 on a scale of 0 to 4), with the mean global improvement rated at 2.6. There were no serious adverse side effects and only two patients noted exacerbation of their tics. Deprenyl appears to be a safe and effective treatment of ADHD in patients with Tourette's syndrome.
I can attest to deprenyl greatly decreasing my ADD symptoms. It would seem the more dopamine you have the more the neuron or neurons need to uptake. It would be like a straw sucking up a pool of dopamine. I don't know if thats exactly true but i have taken deprenyl at 5mgs a day and the effect never stopped after taking it for a week and then so on. There seems to be more to the picture. And of course theres the whole theory of increased theta=delta waves and lower beta waves in the frontal cortex areas. This could be mediated by the low dopamine or some other unknown factor.
As posted earlier, L-DOPA, at least in pharmaceutical doses, has the risk of causing movement issues such as hyperkinesias. I doubt this plays out if supplementing with fava or velvet beans which have generous amounts of L-Dopa.and im just going to take Shepard's word that it doesn't really effect GH