Well, even if you do get higher LDL, it won't oxidize as easily. Oxidized LDL will clog your arteries, but absent oxidation, it is just another part of the symphony of life.
So what they are saying is that hepatic lipid peroxidation reduces LDL secretion. But we know that the liver will produce LDL anyway, with/without anti-oxidants. The fate of the LDL, in a body deficient in Total Anti-oxidant Status, is to be oxidized, then it will accumulate in the artery walls, causing plaque, obstruction, ishcemia, blockage, disfigurement/disease/disabiltity/death.
So, by some twisted logic they say that we should look for anti-oxidants that don't enter the liver, as if ApoB100 were the only molecule sensitive to oxidation/reduction chemistry. There are so many reasons to have high anti-oxidants in the liver that their suggestion is ridiculous. Truly an absurd piece of reasoning.
here is the full study
http://www.pubmedcen...bmedid=15124019