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The calorie fallacy


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#181 tunt01

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Posted 28 March 2010 - 03:50 PM

Animal fat is profoundly important in our health. I once seriously restricted fat intake and became seriously ill. Doctors couldn't figure out why and pills after pills doctors gave me made me worse. What finally cured me was Mary Enig's fat diet. Eat lots of animal fat. Fat does body good.


why specifically animal fat? why not nuts, olive oil/avocado, etc. ?

#182 blackbox

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Posted 28 March 2010 - 03:57 PM

There is an article written by an MIT professor on this issue. He explains why and how our body prefers and requires animal fat. Sorry I don't have url.

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#183 e Volution

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Posted 29 March 2010 - 12:11 AM

There is an article written by an MIT professor on this issue. He explains why and how our body prefers and requires animal fat. Sorry I don't have url.

Yes but this could easily be lumping all plant fats together with seed derived PUFA oils which is obviously drastically different to say EVOO & EVCO. Without seeing it is hard to know; please do try track down that article if you could. I would be interested in reading it as I have also seen hints of the idea before, that animal fatty acids are preferred (or optimal), the question is- is it a net-positive over plant fats which typically are 'cleaner' than animal fats in regards to toxins/unwanted minerals and that animal foods are typically heated before eating...

#184 Sillewater

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Posted 25 May 2010 - 07:28 PM

Training the Obese Beginner Part 2

Lyle Mcdonald, as usual, writes a very good review on training obese beginners. What I wanted to highlight was one paragraph:

TEF can be cut in half due to insulin resistance for example. On a 3000 cal/day diet, for example, where you’d predict TEF to be 300 calories (10%), it might be cut to 150 cal in someone with insulin resistance; which I noted in Part 1 is common in obesity. While this isn’t a massive effect, it does add up over time.


Is this the MAD of low-carb diets over high carb diets?

For a review of metabolic rate: here.

So according to Lyle there are 3 components to metabolic rate
1. Resting Metabolic Rate (which is 70%)
2. Thermic Effect of Food
3. Thermic Effect of Activity

Eades always says its around 250 calories, and looking at the spread in some studies it does seem to be able to get up to 250.

Reprod Nutr Dev. 1996;36(4):391-7.
Thermic effect of food and sympathetic nervous system activity in humans.
Tappy L.

Institute of Physiology, University of Lausanne, Switzerland.
Abstract
The intake of nutrients is known to increase energy expenditure. Measured thermic effects of nutrient are 0-3% for fat, 5-10% for carbohydrates and 20-30% for proteins. Stimulation of adenosine triphosphate (ATP) hydrolysis during intestinal absorption, initial metabolic steps and nutrient storage are responsible for this food thermic effect. The autonomic nervous system modulates the thermic effect of nutrients. Parasympathetic muscarinic antagonists reduce the thermic effect of orally administered nutrients, most likely by delaying gastric emptying and decreasing the amount of nutrient storage. Antagonists of the beta-adrenoreceptors decrease the thermic effect of glucose. The part of glucose induced thermogenesis which is eliminated by beta-adrenergic antagonists has been called 'facultative thermogenesis' and takes place, at least in part, in skeletal muscle. Insulin-induced stimulation of muscle sympathetic nerve activity may be involved in this facultative thermogenesis. The thermic effect of food is reduced in obese, insulin-resistant patients. The effect of thermogenesis represents about 50-150 kcal/day in such patients, and can explain only a minor part of their excess body weight. Defective thermogenesis may, however, contribute to weight gain, or impair weight loss in such patients.

PMID: 8878356 [PubMed - indexed for MEDLINE]



#185 Kristjan

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Posted 26 May 2010 - 09:25 AM

On a high-carb diet insulin is elevated and glucagon is lowered.

On a low-carb diet insulin is lowered and glucagon is elevated.

Insulin and glucagon are the main metabolic hormones, playing a constant tug-of-war between the absorbtive and the postabsorbtive state.

Lowered insulin/elevated glucagon turns on a massive set of catabolic hormones. It seems logical that metabolism would be elevated on a low-carb diet, though maybe "elevated" wouldn't be the right phrase but rather seeing metabolism as being slighly depressed on a high-carb diet, seeing as how the low-carb one is more natural for humans.

#186 Sillewater

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Posted 31 May 2010 - 10:53 PM

Immunobiology. 2009 May 19. [Epub ahead of print]
Endocannabinoids, FOXO and the metabolic syndrome: Redox, function and tipping point - The view from two systems.
Nunn AV, Guy GW, Bell JD.

Metabolic and Molecular Imaging Group, MRC Clinical Sciences Centre, Hammersmith Hospital, Imperial College, Du Cane Road, London W12 OHS, UK.

Two important points arise from this. The first is that the ECS is clearly involved in altering cellular redox, and that this may tie in with FOXO and mitochondrial function, both of which are involved in appetite control. The second suggests that orexigenic circuits may well rely on lower levels of redox to function, whereas anorexic ones rely on higher levels. This would fit with a very basic piece of natural selection: not eating is a Darwinian impossibility. Hence, if you over-eat, especially high carbohydrate meals, the anorexic circuit must eventually fail or compensate to protect itself (e.g. apoptosis, or develop resistance to the excessive signalling), leaving the orexigenic one intact (as it may have stronger anti-ROS systems). This would support the concept that eating is inflammatory and induces oxidative stress (Dandona et al. 2005). It would also support the use of low carbohydrate diets, which can often reverse many symptoms of the metabolic syndrome (Volek and Feinman 2005). It also possibly explains why the anorexic effects of rimonabant are lost so quickly: it is accentuating/inducing stress in the brain, which automatically defaults to the only possible evolutionary strategy of maintaining appetite. This is tightly coupled to resistance to oxidative stress.



#187 DukeNukem

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Posted 01 June 2010 - 01:41 AM

Yeah, Mark Sisson is very badly photo-aged. Can't really speculate on Scott Miller because I have never seen a large photo of him.

If you're on facebook friend me and look at my pics:
http://www.facebook.com/Mi11er

(I'm 49.)

#188 hypnotoad

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Posted 01 June 2010 - 10:14 PM

Yeah, Mark Sisson is very badly photo-aged. Can't really speculate on Scott Miller because I have never seen a large photo of him.



But wasn't Sisson eating a traditional/high carb diet most of his life? I read his post a while back at his blog and there were tons of pics of him a a marathoner all through adulthood. Probably got a lot of sun exposure from it as well.

I think sun and genetics and smoking and stress is way more important than what you have been eating for the past 20 years in terms of appearing old or young.

That brings up another tangential points I thought of watching an Aubrey De Grey you tube clip last night- A few interesting tidbits he mentioned:

1) He doesn't think CR is going to have much effect on humans who are a long-lived species. I assume he means that, ceterus parabis, CR on a person with a generally healthy diet and lifestyle will not change much vs a person who only lives a healthy lifestyle and diet but does no CR. I would imagine even De Grey would find value in CR to offset a typical person's crappy SAD diet though simple mechanisms such as autophagy etc. which are not related to something more exotic/speculative like Sirtuin 1 expression. Even if CR doesn't help greatly extend lifespan, I don't see how it hurts unless you do it wrong.

2) He said he doesn't really do much in his own life to extend his own personal lifespan, and any youthful appearance he may or may not have (despite his beard) has to do with lucky genetics. I find this fascinating as compared with a guy like Kurzweil and others who take every last step they can think off. I'm more in in the Kurzweil camp the older I get, and excellent genetics combined with careful nutrition and "safe" lifestyle (ie not driving, living in a dangerous neighborhood etc) is the ideal combination to make it to 100+

Edited by hypnotoad, 01 June 2010 - 10:16 PM.





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