As has been previously stated, it is not contingent that a paleo diet be high fat / low carbohydrate. Similar to the vegan or vegetarian labels, the paleo label identifies what foods are excluded. Specifically that means no dairy, legumes, grains and processed foods. Within paleo, there are various groups that utilize vastly different macro-nutrient ratios. TheFountain: it seems you have a problem with is low-carb diets. I recall you mentioning you had issues with a vegetarian low-carb as well. You have a problem with dietary fat, not paleo.
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I don't know why TheFountain can't handle dietary fat. Maybe genetics? He's probably APOE4/4 or something. I'm APOE3/3 and my body does just fine on low-carb and low-fat. If TheFountain does have a APOE4 geneotype, it is probably good that he eats low fat as dietary fat will cause him to have advanced CVD at a young age.
What if it is the inclusion of meat that increases testosterone and also lack of carbohydrate that increases testosterone (keeping abdominal fat at bay -- Meat eating paleo vs vegetarian/vegan/plant/phytoestrogen paleo should be different).
GoodCalories, BadCalories (2007) on LPL and hormone testosterone:
http://paste2.org/p/1430577
[line 309]
Insulin, not surprisingly, is the primary regulator of LPL activity, although not the only one. This regulation functions
differently, as is the case with all hormones, from tissue to tissue and site to site. In fat tissue, insulin increases LPL
activity; in muscle tissue, it decreases activity. As a result, when insulin is secreted, fat is deposited in the fat tissue,
and the muscles have to burn glucose for energy. When insulin levels drop, the LPL activity on the fat cells decreases
and the LPL activity on the muscle cells increases—the fat cells release fatty acids, and the muscle cells take them up
and burn them.
It’s the orchestration of LPL activity by insulin and other hormones that accounts for why some areas of the body will
accumulate more fat than others, why the distribution of fat is different between men and women, and how these
distributions change with age and, in women, with reproductive needs. Women have greater LPL activity in their
adipose tissue than men do, for example, and this may be one reason why obesity and overweight are now more
common in women than in men. In men, the activity of LPL is higher in the fat tissue of the abdominal region than in
the fat tissue below the waist, which would explain why the typical male obesity takes the form of the beer belly.
Women have more adipose-tissue LPL activity in the hips and buttocks than in the abdominal region, although after
menopause the LPL activity in their abdominal region catches up to that of men.
These various fat deposits are also regulated over time by the changing flux of sex hormones, so LPL can be
considered the point at which insulin and sex hormones interact to determine how and when we fatten. The male sex
hormone testosterone, for instance, suppresses LPL activity in the abdominal fat, but has little or no effect on the LPL
in the fat of the hips and buttocks. Increasing fat accumulation in the abdomen as men age may therefore be a product
of both increasing insulin and decreasing testosterone. The female sex hormone progesterone increases the activity of
LPL, particularly in the hips and buttocks, but estrogen, another female sex hormone, decreases LPL activity.*120 It’s
the decrease in estrogen secretion during menopause—and so the increase in LPL activity—that may explain why
women frequently gain weight as they pass through menopause. The effect of decreasing estrogen secretion on LPL
activity would also explain why women typically fatten after the removal of the uterus in a hysterectomy. The change
in hormonal regulation of LPL also explains how and why fat deposition changes during pregnancy and, after birth,
with nursing.
and from a recent 2009 review on LPL
http://ajpendo.physi...297/2/E271.long
Growth hormone and sex steroid hormones such as testosterone and estrogen inhibit adipose tissue LPL activity and promote lipid mobilization (reviewed in Ref. 23) but increase heart and skeletal muscle LPL activities (181). The effect of these hormones is believed to be mediated by the androgen receptor, the density of which is higher in visceral than subcutaneous adipose tissue. In sedentary obese men, plasma testosterone and bioavailable testosterone levels are inversely correlated with femoral and abdominal wall adipose tissue LPL (207). Testosterone treatment of abdominally obese men also produces a decrease in visceral fat mass (216).
...
LPL activity has been reported to increase as a function of fat cell size (22, 57, 89, 230, 241), and sex differences seem to alter the relationship between LPL activity and fat cell size. Fat cell size is greater in females than males in the thigh and gluteal regions but not in the abdomen. Fasting LPL activity/fat cell correlates well with the fat cell size in females in all three areas, but only in the abdomen and thigh in men (270). This may relate to the fundamental differences in the regulation of TG uptake between males and females in different regions of adipose tissue.
Sidenote: After reading that... maybe physiologically, men have more to benefit in fat loss with high animal, testosterone diets than women. Also sidenote: women are less likely to adhere to low-carb diets:
http://www.socresonl....uk/16/2/8.html
I share the concerns of both these groups of critics, although both the obesity epidemic and low-carbohydrate movement have arguably weakened the association of dieting with women. Concern about obesity has shifted weight-loss coverage in the media away from women specifically (Boero 2007: 44n1), and low-carbohydrate diets seem to appeal particularly to men (Bentley 2005), with at least one survey showing that men are much more likely than women to follow a low-carbohydrate diet long-term (Blanck et al. 2006). Low-carbohydrate diets forego ‘appearance claims’ in favour of urgent warnings about the dangers of the modern Western diet (Mouton 2001: para 19). In this article I therefore take a somewhat different approach, examining the particular form of the cultural and discursive response to the obesity epidemic represented by the low-carbohydrate diet movement. I situate my critique in relation to previous work in sociology and food studies on themes of nostalgia and cultural identity (eg Pickering & Keightley 2006; Valentine 1999), which are especially relevant topics in the context of dietary change, both individual and cultural.
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You're such a fucking drama queen. Get a fucking social life dude, you're too fucking young to be spending this kind of time arguing such shit on the internet.
Hey thanks for the advice.
But i've been exercising pretty hard as of late to compensate for all this drama! Exercise is the anti-drama, ya see.
Also over over-exercise training is linked to high cortisol.
Robbwolf and his partners on podcast talk about gaining belly fat from their own overtraining all the time (listen to the older podcast sessions to hear it).
Quote from ThePaleoSolution 2010 book
Cortisol raises blood sugar levels, which can cause fat gain. Although many people don’t know this, cortisol release from stress and a lack of sleep
factors prominently in body fat gain, leading to that pesky spare tire around the midsection. Cortisol shouldn’t be feared, because it is a crucial antiinflamatory—
we just don’t want too much of it.
____________________________
Geek-Speak
Cortisol is often referred to as a “stress hormone,” given that it is released in response to stress and anxiety. Cortisol increases blood pressure and acts
as an anti-inflammatory by lowering the activity of the immune system. It will trigger the breakdown of muscle mass by converting protein (amino acids)
into glucose via gluconeogenesis. Cortisol decreases insulin sensitivity, lowers the rate of bone formation, and causes a loss of collagen in the skin and
other connective tissues. The following increase cortisol levels: intense or prolonged physical activity, caffeine, sleep deprivation, stress, subcutaneous fat
tissue, and certain contraceptives.
Sidenote: EdNorton (from Fightclub fame) recently admitted to gaining weight in other areas from chronic exercise with the Masai
http://www.youtube.c...u/0/F1p8Ttb3q4U
Edited by hggh, 21 September 2011 - 09:13 AM.