I've largely answered my own question here. I am using both Cantrophenoxine (Lipofuscin removal) and Rosmarinic Acid (an attempt at AGE crosslink removal) for 2 months PRIOR, then waiting an additional month before doing this stem cell/senolytic protocol. That way I separate them out so that the two do not interfere with each other and potentially cause problems. I'll see what happens.
Would it be reasonable to say that the stem cell/senolytic protocol can aid in or be a form of ECM turnover process? Given that every cell in our bodies come from a stem cell and that this would presumably include the fibroblasts, that renewing one's fibroblasts ought to assist in ECM turnover, thus helping to reduce or eliminate AGE crosslinks.
